This paper investigates the phenomenon of age-related chronoperceptual acceleration—the subjective experience that time passes more quickly as one ages—and proposes a metabolic model to explain its biological underpinnings. Survey data from 21 participants demonstrate that accelerated time perception is a near-universal experience among adults, with 95.2% of respondents reporting this phenomenon. The study advances a mechanistic explanation grounded in hippocampal neurophysiology: age-related decline in mitochondrial ATP production leads to increased GABAergic inhibitory activity, which compromises episodic memory encoding along the temporo-septal axis. This reduction in episodic memory density results in retrospective temporal compression—fewer encoded experiences within a given interval translate to the perception that time moved rapidly. The model integrates the GABAergic deafferentation hypothesis with episodic memory research to provide a biophysiological framework for understanding chronoperceptual changes across the lifespan. Implications for early diagnosis of neurodegenerative diseases and therapeutic interventions are discussed.